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2001. Occupational stress series. Heart disease.

May 25, 2012
by Andrew@Reliabilityoxford.co.uk
0 Comment
A causal link between stress and heart disease would prompt significant liability exposure. Heart disease is very common. Feelings of being hassled are very common, and may be caused by heart disease or high blood pressure. Unravelling this knot will draw on disparate and incomplete studies.

Evidence from:

T.Hallman et al. Journal of Cardiovascular Risk Feb (2001) Vol.8 #1 p.39.

After suffering manifest coronary disease it was found that compared with controls, there was excess family strife, physical stress, burn-out, daily hassles. Some of these could be subject to modification at work.

The Radar report is available to subscribers:

SK 1#2 7

Evidence from:

GW Evans et al. Journal of Applied Psychology. Oct (2000) Vol. 85 #5 p.779.

40 experienced female office workers were assigned at random to a well-controlled trial of office work with or without open office noise at 55 dBA.

Epinephrine, nor epinephrine and cortisol were measured before and after the test period. Subjects were asked to solve insoluble problems after the test period.

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SK 1#3 5

Evidence from:

CK Nordstrom et al. Epidemiology.  March (2001) Vol. 12 #2 p.180.

The authors claim that raised lesions in the left and right carotid arteries were more common or more pronounced in the higher stress groups after 18 months. The effect was confined to men.

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SK 1#3 8

Evidence from:

A Steptoe. Journal of Psychosomatic Research  (2001) Vol. 50 #2  p 57.

One interpretation would be that chronic low control may be harmless, but that unfamiliar episodes of low control may not be.

It is not clear whether or not the perception of low control while at work is enhanced by a perceived change in blood pressure. If it were, the study may have been over-interpreted.

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SK 1#4 4

Evidence from:

A Tsutsumi et al. Occupational and Environmental Medicine. June (2001) Vol.58 #6 p 367.

Hypertension was associated with age, family history and body mass index (BMI). There was a barely detectable association with job strain (high demands + low control).

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SK 1#5 5

Evidence from:

S Levenstein et al. Arch Intern Med. May (2001) Vol. 28;161(10) p 1341-6.

Psychosocial stressors have been shown to predict hypertension in several cohort studies.

In this study, 2357 adults, free of hypertension in 1974 were surveyed at baseline for psychosocial, behavioral, and socio-demographic factors.

20 years later 637 reported having ever used anti-hypertensive medication (27.9% of the men and 26.3% of the women).

In multivariate models, job insecurity (odds ratio, 1.6), unemployment (odds ratio, 2.7), and low self-reported job performance (odds ratio, 2.1) remained independent predictors of hypertension in men, whereas low-status work (odds ratio, 1.3) was an independent predictor of hypertension in women.

Comment
Given that these factors could all change in space of a few months, it would have been of interest to monitor the baseline data at repeated points during the 20-year study period.

Unemployment was the most predictive risk factor.

Evidence from:

RS Vasan et al. The Lancet. November (2001) Vol. 358 #9294 p 1682.

Stress researchers hope to link changes in blood pressure to work conditions. The research here provides baseline data on the normal working population and how blood pressure changes with age. Stress would have to measureably add to normal variation.

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SK 1#11 4

Evidence from:

M Kivimaka et al. Work and Stress. March (2001) Vol. 15#1 p 3.

The authors concluded that shift work leads to bad health habits, assuming initial equivalence has
always been the case (which they do not know).

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SK 1#12 6

Evidence from:

TM Pollard. Work and Stress. March (2001) Vol. 15#1 p. 14.
In theory, tenure uncertainty before a workplace reorganisation and role uncertainty afterwards should be associated with changes in anxiety, blood pressure and other indicators of well being.
98 women and 86 men were checked at 6-month intervals twice before and twice after a fundamental workplace reorganisation. None were diabetic or existing coronary heart disease cases. 196 out of 282 volunteers survived the changes and were reassessed at stages 3 and 4.
Tense arousal peaked just before change, mental health dipped, BMI remained constant as did BP (averages for systolic and diastolic were 120 and 76 mmHg), and cholesterol.
Changes to tense arousal and mental health were barely statistically significant.
Comment
Although the authors claim significant changes occurred, these were not immediately evident from the results. No measurement was made of those who left at the time of the reorganisation.
Even if changes had been detected, their relationship with diagnosable harm is indirect. Foreseeability of harm at times of change is not strongly supported by this research.

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